Kaffe + Insulinpåslag

[praktsvinet]

Som jag har förstått det så ger kaffet i sig inget nämnvärt insulinpåslag. Men om kaffet intas i samband med måltid kan det höja insulinpåslaget på den måltiden med upp till 60%?

Min fråga är då hur man lättast minimerar detta insulinpåslag. Den första tanken som slår mig är ju att man helt enkelt väntar med kaffet, men hur länge bör man vänta? Och hjälper det verkligen att vänta? Eller finns det något annat sätt?

[King Grub]

Varför tror du att du påverkas negativt av det?

[praktsvinet]

Varför tror du att du påverkas negativt av det?

Jag försöker hålla mitt insulinpåslag så låg/jämn nivå som möjligt. Då det sägs påverka dom androgena nivåerna i blodet, som i sin tur kan ge upphov till dålig hy.

Jag är därför nyfiken på om detta är möjligt.

[Ola Wallengren]

Lite varierande resultat men hur som helst så är det inget att oroa sig för. Kaffe minskar ju risken för typ2 diabetes så hur illa kan det va.

Eur J Nutr. 2011 Oct 25. [Epub ahead of print]
Coffee does not modify postprandial glycaemic and insulinaemic responses induced by carbohydrates.
Hätönen KA, Virtamo J, Eriksson JG, Sinkko HK, Erlund I, Jousilahti P, Leiviskä JM, Valsta LM.
SourceDepartment of Lifestyles and Participation, National Institute for Health and Welfare, Mannerheimintie 166, 00271, Helsinki, Finland, katja.hatonen@thl.fi.

Abstract
BACKGROUND: Strong epidemiological evidence suggests that coffee consumption is associated with lower risk of type 2 diabetes. In postprandial studies, however, caffeine consumption has been associated with impaired glucose regulation.

AIM OF THE STUDY: To study the acute effects of coffee and caffeine-containing soft drinks on glycaemic and insulinaemic responses.

DESIGN: Twelve healthy volunteers were served each test food once and the reference glucose solution twice, containing 50 g of available carbohydrates, after an overnight fast at 1-week intervals in a random order. Capillary blood samples were drawn at 15-30 min intervals for 2 h after each study meal. The incremental areas under the curve (IAUC), glycaemic index (GI) and insulinaemic index (II), were calculated to estimate the glycaemic and insulinaemic responses.

RESULTS: Glucose and insulin responses of coffees with glucose containing 150 or 300 mg of caffeine did not differ from responses of pure glucose solution; the GIs were 104 and 103, and the IIs were 89 and 92, respectively. When a bun or sucrose and milk were consumed together with coffee, lower GI values and insulin responses were observed, reflecting the carbohydrate quality and protein content of the accompaniments. Sucrose-sweetened cola produced a high GI value of 90 and an II of 61.

CONCLUSIONS: Coffee does not modify glycaemic and insulinaemic responses when ingested with a carbohydrate source. Therefore, there is no need to avoid coffee as a choice of beverage in GI testing.



Consumption of caffeinated coffee and a high carbohydrate meal affects postprandial metabolism of a subsequent oral glucose tolerance test in young, healthy males.
Moisey LL, Robinson LE, Graham TE.
SourceDepartment of Human Health and Nutritional Sciences, University of Guelph, Guelph, ON, Canada.

Abstract
Caffeine and caffeinated coffee (CC) elicit acute insulin insensitivity when ingested before a carbohydrate load. The effects of CC on glucose tolerance and insulin sensitivity when co-ingested with a high carbohydrate meal and on postprandial metabolism of a subsequent (second) carbohydrate load have not been studied. In a randomised, crossover design, ten healthy males ingested either CC (5 mg caffeine/kg body weight), decaffeinated coffee (DC) or water (W; equal volume) co-ingested with a high glycaemic index cereal followed 3 h later by a 75 g oral glucose tolerance test. After the initial meal, insulin area under the curve (AUC) and insulin sensitivity index did not differ between treatments, although glucose AUC for CC (107 (sem 18) mmol/l x 3 h) and DC (74 (sem 15) mmol/l x 3 h) was greater than W ( - 0.2 (sem 29) mmol/l x 3 h, P < 0.05). After the second carbohydrate load, insulin AUC for CC was 49 % and 57 % greater (P < 0.01) than for DC and W, respectively. Despite the greater insulin response, glucose AUC for CC (217 (sem 24) mmol/l x 2 h) was greater than both DC (126 (sem 11) mmol/l x 2 h, P = 0.01) and W (55 (sem 34) mmol/l x 2 h, P < 0.001). Insulin sensitivity index after the second meal was lower after CC (8.2 (sem 0.9)) compared with both DC (12.4 (sem 1.2), P < 0.01) and W (13.4 (sem 1.4), P < 0.001). Co-ingestion of CC with one meal resulted in insulin insensitivity during the postprandial phase of a second meal in the absence of further CC ingestion. Thus, CC may play a role in daily glycaemic management.



Am J Clin Nutr. 2008 May;87(5):1254-61.
Caffeinated coffee consumption impairs blood glucose homeostasis in response to high and low glycemic index meals in healthy men.
Moisey LL, Kacker S, Bickerton AC, Robinson LE, Graham TE.
SourceDepartment of Human Health and Nutritional Sciences, University of Guelph, Guelph, Ontario, Canada.

Abstract
BACKGROUND: The ingestion of caffeine (5 mg/kg body weight) and a 75-g oral glucose load has been shown to elicit an acute insulin-insensitive environment in healthy and obese individuals and in those with type 2 diabetes.

OBJECTIVE: In this study we investigated whether a similar impairment in blood glucose management exists when coffee and foods typical of a Western diet were used in a similar protocol.

DESIGN: Ten healthy men underwent 4 trials in a randomized order. They ingested caffeinated (5 mg/kg) coffee (CC) or the same volume of decaffeinated coffee (DC) followed 1 h later by either a high or low glycemic index (GI) cereal (providing 75 g of carbohydrate) mixed meal tolerance test.

RESULTS: CC with the high GI meal resulted in 147%, 29%, and 40% greater areas under the curve for glucose (P < 0.001), insulin (NS), and C-peptide (P < 0.001), respectively, compared with the values for DC. Similarly, with the low GI treatment, CC elicited 216%, 44%, and 36% greater areas under the curve for glucose (P < 0.001), insulin (P < 0.01), and C-peptide (P < 0.01), respectively. Insulin sensitivity was significantly reduced (40%) with the high GI treatment after CC was ingested compared with DC; with the low GI treatment, CC ingestion resulted in a 29% decrease in insulin sensitivity, although this difference was not significant.

CONCLUSION: The ingestion of CC with either a high or low GI meal significantly impairs acute blood glucose management and insulin sensitivity compared with ingestion of DC. Future investigations are warranted to determine whether CC is a risk factor for insulin resistance.

[Ola Wallengren]

Jag försöker hålla mitt insulinpåslag så låg/jämn nivå som möjligt. Då det sägs påverka dom androgena nivåerna i blodet, som i sin tur kan ge upphov till dålig hy.

Jag är därför nyfiken på om detta är möjligt.

Visst, teoretiskt sett verkar det finnas en koppling men i praktien är det viktigare vad och hur mycket du äter tillsammans med kaffet, och vid alla andra tidpunkter med för den delen.
Första studien pekar ju på att Insulinindex inte förändras av kaffe.

[praktsvinet]

Tack för snabba svar.

Jag konsumerar ju inga större mängder kaffe per dag, max 3 koppar samt håller i övrigt en bra kost. Så detta kanske inte är något jag behöver oroa mig över. Och som jag förstår det
av den första studie kan jag t.o.m. med fördel även ha mjölk i mitt kaffe (i mitt fall sojamjölk).

[King Grub]

Så detta kanske inte är något jag behöver oroa mig över.

Nej, inte alls.