Hypertrofiinriktad styrketräning minskar inflammationer och bukfett

[King Grub]

PURPOSE:

Our primary aim was to study the effects of a 4 wk preparatory resistance training (RT) period followed by 12 wks of two specific RT protocols (either hypertrophic-strength, HS, or strength-hypertrophy-power, SHP, training) on inflammation markers and the possible relationship of the changes in abdominal fat and lean mass to the changes in inflammation status.

METHODS:

A total of 82 healthy men were included in the study. Maximal concentric leg press strength (1RM), total body lean mass, total body and abdominal fat mass, circulating high-sensitivity C-reactive protein (hs-CRP), interleukin-6 (IL-6), interleukin-1 receptor antagonist (IL-1ra), monocyte chemoattractant protein 1 (MCP-1), and selected adipocytokines (resistin, adiponectin and leptin) concentrations were measured before (PRE) and after 4 (wk4) and 16 weeks (wk16) of RT.

RESULTS:

After the initial phase of RT, on wk4, abdominal and total fat mass as well as plasma leptin concentrations were significantly reduced (p<0.05), whereas muscle mass, IL-1ra, resistin and MCP-1 concentrations were significantly increased (p<0.05). During specialized training phase, at wk16, only HS led to further reduction in abdominal and total fat mass, resistin, and leptin (p<0.05), whereas both training modes led to lower MCP-1 concentrations (p<0.05).

CONCLUSION:

Abdominal fat mass and circulating leptin were reduced already after 4 wks of RT. Simultaneously, circulating MCP-1 and resistin concentrations increased, possibly as markers of muscle adaptation and regeneration. The present findings also suggest that RT with hypertrophic focus is beneficial for further reductions in abdominal fat mass and to decrease circulating inflammatory markers.

Appl Physiol Nutr Metab. 2017 Oct 20. INFLAMMATION STATUS OF HEALTHY YOUNG MEN: INITIAL AND SPECIFIC RESPONSES TO RESISTANCE TRAINING.