Vad heter tårna?

[Krokodil]

Vad heter de tre tårna mellan lilltån och stortån?

[King Grub]

Tobele, Makafru och Spelaros. :D

[Osten80]

Fråga Stig Helmers morsa, hon vet :D

[mela]

pekfingertån, långfingertån och ringfingertån :MrT: :D

[pumpare]

pekfingertån, långfingertån och ringfingertån :MrT: :D

Vad kallar du de tre fingrarna mellan tummen och lillen då? pektåfingern, långtåfingern och ringtåfingern? :D

[Princy]

Tobele, Makafru och Spelaros. :D

Vad är det där för namn? :laugh:

[mela]

Vad kallar du de tre fingrarna mellan tummen och lillen då? pektåfingern, långtåfingern och ringtåfingern? :D
nej har ju döpt tårna efter fingrarna..säger man somjag sa så fattar alla :D

[King Grub]

Vad är det där för namn? :laugh:

Titele, Tobele, Makafru, Spelaros och Stortampen sade vi alltid när jag var liten.

[Bronkitkorv]

Tilltå, långeman, Lilltå tror jag !

[z_bumbi]

Hittade dessa

"Stortå, tilletå, timpen, tampen och lilltyta."


"Tummetott, slickepott, långeman, gullebrand och lilla vickevire." <-- Den känner jag igen.

"Rött är sött - sa tummetott
Blått är flott - sa slickepott
Grönt är skönt - sa långeman
Gult är fult - sa gullebrand
Det tycker inte jag - sa lilla vickevire."

Jag röstar för att den med tummetott osv gäller fingrar..

[King Grub]

"Tummetott, slickepott, långeman, gullebrand och lilla vickevire." <-- Den känner jag igen.

Det är ju fingrarna.

[Slartibartfast]

Denhär tråden är inte speciellt macho. :MrT:

[z_bumbi]

Det är ju fingrarna.

Japp. Men jag är inte så snabb på att kutta och pasta.

[King Grub]

Japp. Men jag är inte så snabb på att kutta och pasta.

Du postar för få obegripliga forskningsrapporter som ingen läser, men som ser imponerande ut och som ger tyngd åt ens svammel.

[Sheogorath]

Pektån, långtån och ringtån, så klart :MrT:.

[reinholdsson]

Tilltå, långeman, Lilltå tror jag !

Vad heter den minstra tån då? mikrotån? :D

[aliquis]

Du postar för få obegripliga forskningsrapporter som ingen läser, men som ser imponerande ut och som ger tyngd åt ens svammel.Ah, det är så man blir KUNG!

"Direct Asymmetric Intermolecular Aldol Reactions Catalyzed by Amino Acids and Small Peptides.

Cordova A, Zou W, Dziedzic P, Ibrahem I, Reyes E, Xu Y.

Department of Organic Chemistry, The Arrhenius Laboratory, Stockholm University, 106 91 Stockholm, Sweden, Fax: (+46) 8-154-908.

In nature there are at least nineteen different acyclic amino acids that act as the building blocks of polypeptides and proteins with different functions. Here we report that alpha-amino acids, beta-amino acids, and chiral amines containing primary amine functions catalyze direct asymmetric intermolecular aldol reactions with high enantioselectivities. Moreover, the amino acids can be combined into highly modular natural and unusual small peptides that also catalyze direct asymmetric intermolecular aldol reactions with high stereoselectivities, to furnish the corresponding aldol products with up to >99 % ee. Simple amino acids and small peptides can thus catalyze asymmetric aldol reactions with stereoselectivities matching those of natural enzymes that have evolved over billions of years. A small amount of water accelerates the asymmetric aldol reactions catalyzed by amino acids and small peptides, and also increases their stereoselectivities. Notably, small peptides and amino acid tetrazoles were able to catalyze direct asymmetric aldol reactions with high enantioselectivities in water, while the parent amino acids, in stark contrast, furnished nearly racemic products. These results suggest that the prebiotic oligomerization of amino acids to peptides may plausibly have been a link in the evolution of the homochirality of sugars. The mechanism and stereochemistry of the reactions are also discussed.

PMID: 16637082 [PubMed - as supplied by publisher]"

"Proteomic analysis of the anterior cingulate cortex in the major psychiatric disorders: Evidence for disease-associated changes.

Beasley CL, Pennington K, Behan A, Wait R, Dunn MJ, Cotter D.

Department of Psychological Medicine, Institute of Psychiatry, London, UK.

Abnormalities of the anterior cingulate cortex have previously been described in schizophrenia, major depressive disorder and bipolar disorder. In this study 2-DE was performed followed by mass spectrometric sequencing to identify disease-specific protein changes within the anterior cingulate cortex in these psychiatric disorders. The 2-DE system comprised IPGs 4-7 and 6-9 in the first, IEF dimension and SDS-PAGE in the second dimension. Resultant protein spots were compared between control and disease groups. Statistical analysis indicated that 35 spots were differentially expressed in one or more groups. Proteins comprising 26 of these spots were identified by mass spectroscopy. These represented 19 distinct proteins; aconitate hydratase, malate dehydrogenase, fructose bisphosphate aldolase A, ATP synthase, succinyl CoA ketoacid transferase, carbonic anhydrase, alpha- and beta-tubulin, dihydropyrimidinase-related protein-1 and -2, neuronal protein 25, trypsin precursor, glutamate dehydrogenase, glutamine synthetase, sorcin, vacuolar ATPase, creatine kinase, albumin and guanine nucleotide binding protein beta subunit. All but three of these proteins have previously been associated with the major psychiatric disorders. These findings provide support for the view that cytoskeletal and mitochondrial dysfunction are important components of the neuropathology of the major psychiatric disorders.

PMID: 16637010 [PubMed - as supplied by publisher]"

"Analysis of the SWI4/SWI6 protein complex, which directs G1/S-specific transcription in Saccharomyces cerevisiae.

Sidorova J, Breeden L.

Fred Hutchinson Cancer Research Center, Seattle, Washington 98104-2092.

SWI4 and SWI6 play a crucial role in START-specific transcription in Saccharomyces cerevisiae. SWI4 and SWI6 form a specific complex on the SCB (SWI4/6-dependent cell cycle box) sequences which have been found in the promoters of HO and G1 cyclin genes. Overproduction of SWI4 eliminates the SWI6 dependency of HO transcription in vivo and results in a new SWI6-independent, SCB-specific complex in vitro, which is heterogeneous and reacts with SWI4 antibodies. The C terminus of SWI4 is not required for SWI6-independent binding of SWI4 to SCB sequences, but it is necessary and sufficient for association with SWI6. Both SWI4 and SWI6 contain two copies of a 33-amino-acid TPLH repeat, which has been implicated in protein-protein interactions in other proteins. These repeats are not required for the SWI4-SWI6 association. Alanine substitutions in both TPLH repeats of SWI6 reduce its activity but do not affect the stability of the protein or its association with SWI4. However, these mutations reduce the ability of the SWI4/6 complex to bind DNA. Deletion of the lucine zipper motif in SWI6 also allows SWI4/6 complex formation, but it eliminates the DNA-binding ability of the SWI4/6 complex. This indicates that the integrity of two different regions of SWI6 is required for DNA binding by the SWI4/6 complex. From these data, we propose that the sequence-specific DNA-binding domain resides in SWI4 but that SWI6 controls the accessibility of this domain in the SWI4/6 complex.

PMID: 8423776 [PubMed - indexed for MEDLINE]"

"Presymptomatic genetic diagnosis for consulters from a large Chinese family with juvenile open angle glaucoma.

Chen Y, Jiang D, Wan B, Yu L, Sun X.

State Key Laboratory of Genetic Engineering, Department of Ophthalmology and Vision Science, Eye and ENT Hospital, Institute of Genetics, School of Life Sciences, Fudan University, Shanghai, China.

PURPOSE: To provide genetic counseling and presymptomatic diagnosis to family members by investigating the genetic cause of a large primary juvenile open angle glaucoma (JOAG) pedigree with an autosomal dominant pattern. METHODS: Ocular examinations were performed on all members of the pedigree in order to determine their disease status. Subjects were labeled as affected individuals, unaffected individuals, and suspects. Genomic DNA was extracted from the peripheral blood of the family members. Three exons of MYOC, 12 coding exons of OPTN, and two coding exons of CYP1B1 were amplified by polymerase chain reaction (PCR). Direct DNA sequencing was used to identify mutations in MYOC, OPTN, and CYP1B1. Presymptomatic diagnoses were made for the consulters based on the results of both clinical examination and genetic analysis. RESULTS: One heterozygous mutation in the MYOC gene was identified in all patients of the pedigree. It was a cytosine to thymine transition at nucleotide 1,109, which corresponded to an amino acid residue change from proline to leucine at codon 370. The Pro370Leu mutation correlated with a severe JOAG phenotype as previously reported. Two adolescents, who were labeled as suspects, were detected to carry the same mutation and thus had a high risk of developing glaucoma. Close follow-up at regular intervals was recommended. In addition, no pathogenic mutations of OPTN and CYP1B1 were detected. CONCLUSIONS: The Pro370Leu mutation of the MYOC gene contributed to JOAG in this pedigree. Early onset, rapid development, poor response to medicine and high penetrance are characteristics of this mutation. Genetic analysis is valuable for providing genetic counseling and presymptomatic diagnosis to members in typical autosomal inheritance pedigrees with JOAG."

"Supplementation with omega-3 polyunsaturated fatty acids augments brachial artery dilation and blood flow during forearm contraction.

Walser B, Giordano RM, Stebbins CL.

Department of Internal Medicine, Division of Cardiovascular Medicine, TB 172, University of California, Davis, CA, 95616-8634, USA.

Omega-3 polyunsaturated fatty acids such as docosahexaenoic acid (DHA) and eicosapentaenoic acid (EPA) have beneficial effects on the heart and vasculature. We tested the hypothesis that 6 weeks of dietary supplementation with DHA (2.0 g/day) and EPA (3.0 g/day) enhances exercise-induced increases in brachial artery diameter and blood flow during rhythmic exercise. In seven healthy subjects, blood pressure, heart rate and brachial artery diameter, blood flow, and conductance were assessed before and during the last 30 s of 90 s of rhythmic handgrip exercise (30% of maximal handgrip tension). Blood pressure (MAP), heart rate (HR), and brachial artery vascular conductance were also determined. This paradigm was also performed in six other healthy subjects who received 6 weeks of placebo (safflower oil). Placebo treatment had no effect on any variable. DHA and EPA supplementation enhanced contraction-induced increases in brachial artery diameter (0.28 +/- 0.04 vs. 0.14 +/- 0.03 mm), blood flow (367 +/- 65 vs. 293 +/- 55 ml min-1) and conductance (3.86 +/- 0.71 vs. 2.89 +/- 0.61 ml min-1 mmHg-1) (P < 0.05). MAP and HR were unchanged. Results indicate that treatment with DHA and EPA enhances brachial artery blood flow and conductance during exercise. These findings may have implications for individuals with cardiovascular disease and exercise intolerance (e.g., heart failure).

PMID: 16636858 [PubMed - as supplied by publisher]"

Källa:
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=pubmed&dopt=Abstract&list_uids=16637082&query_hl=1&itool=pubmed_docsum
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=pubmed&dopt=Abstract&list_uids=16637010&query_hl=3&itool=pubmed_docsum
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=pubmed&dopt=Abstract&list_uids=8423776&query_hl=5&itool=pubmed_docsum
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=pubmed&dopt=Abstract&list_uids=16636654&query_hl=7&itool=pubmed_docsum
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=pubmed&dopt=Abstract&list_uids=16636858&query_hl=9&itool=pubmed_docsum


Ni behöver inte kyssa mina tår, jag är kittlig!

Det är helt ok att ta bort dem, framförallt om det inte är lagligt att göra sådär?

[z_bumbi]

Du postar för få obegripliga forskningsrapporter som ingen läser, men som ser imponerande ut och som ger tyngd åt ens svammel.

Sant, jag ska börja klistra in allt jag har läst senaste veckorna.

Iofs tror jag Larson har någon typ av copyright. :D

[Krokodil]

Denhär tråden är inte speciellt macho. :MrT:
Tvärtom min käre Slarti. Man måste vara jäkligt macho för att kunna starta en sån här tråd utan att skämmas... :D

[Zakath]

Du postar för få obegripliga forskningsrapporter som ingen läser, men som ser imponerande ut och som ger tyngd åt ens svammel.

Jag läser dem! Både intressant + bra engelskaträning :)

[Tjej83]

Lilltå, tilltå, tavvarota, löttlött och storböck. Sådeså! ;)

[z_bumbi]

Lilltå, tilltå, tavvarota, löttlött och storböck. Sådeså! ;)

Klart bästa förslaget.

[dynamo]

Tror vi sa stortå, långtå, mellantå, ringtå, lilltå.. är lite osäker på ringtå.

Efter googling på andra alternativ samt vad jag läst i tråden här lutar jag åt att i framtiden benämna dem som läkare gör, med en liten modifikation. Stortån, andra tån, tredje tån, fjärde tån och lilltån (istället för femte tån).

Kan även enkelt anpassas för de med färre eller fler tår på varje fot!

[n1tro]

det du vill att dom ska heta / ring svenska akademien

[Princy]

Titele, Tobele, Makafru, Spelaros och Stortampen sade vi alltid när jag var liten.

Vad gulligt! Det låter som utomjordingar eller något. :D

[myggan]

Du postar för få obegripliga forskningsrapporter som ingen läser, men som ser imponerande ut och som ger tyngd åt ens svammel.


:laugh:

[Metal_boy_]

Bröderna Dalton.

[kharon]

Stortå, tåtå, tåtilla, tililesa, lillstumpan.


Har jag hört... :em:

[T-train]

Grubens variant har jag också hört, bra den med..annars är min favvo: totå, tåtilla, tillerosa, knoppknosa - och lille virrevasken :)

[christian]

Stortå, tvåtå, melltå, fyrtå, lilltå har jag hittat.