Effektivt med snus vid deff?

[quinx]

Har för mig att jag läst tidigare här på forumet att nikotin ökade ämnesomsättningen. Skulle det då inte vara en idè att köpa en stock snus inför deffen enbart för de egenskaperna?

Det låter nog hyffsat knäppt, men man kan ju fråga ;) :D

[hassezilla]

Har för mig att jag läst tidigare här på forumet att nikotin ökade ämnesomsättningen. Skulle det då inte vara en idè att köpa en stock snus inför deffen enbart för de egenskaperna?

Det låter nog hyffsat knäppt, men man kan ju fråga ;) :D
Blir nog inga märkbara effekter. Inte för mig iaf.

[quinx]

Ok, men jag är ickesnusare så då kanske skillnaden märks? Fast du kanske är ickesnusare du med?

[Frazze]

Du är väl inte allvarlig?

Gå på en tio min längre morgonpromenad istället för att börja "äta" beroendeframkallande och potentiellt cancerogena saker.

Och om du nu absolut tror att nikotinet är ett smart sett att få upp förbränningen så köp nikotinplåster istället. Men det låter otroligt dumt i alla fall.

men ja, en idé är det, om än inte en så smart sådan.

[King Grub]

Med Sci Sports Exerc. 1992 Mar;24(3):389-95. The effects of oral smokeless tobacco on the cardiorespiratory response to exercise.

The purpose of this investigation was to determine the effects of oral smokeless tobacco (OST) usage on oxygen uptake (VO2), cardiac output (Qc), stroke volume (SV), heart rate (HR), and plasma lactate concentration (Lc) during rest and exercise. Fifteen asymptomatic subjects were recruited from 18 to 33-yr-old male users of OST. Comparisons of the responses of VO2, Qc, SV, HR, and Lc were made between 2.5-g OST and placebo experimental conditions during rest and at 60% and 85% maximal VO2 treadmill exercise. Plasma nicotine concentrations (Nc) were determined by radioimmunoassay. There were significant increases in HR and Lc and a decrease in SV during rest and at 60% and 85% maximal exercise (P less than 0.05). Furthermore, there were no significant differences in maximal HR, Lc, and VO2 (P greater than 0.05). In conclusion, these data indicate that the increased Nc incurred by OST usage increases anaerobic energy production and produces an increased tachycardiac response to a given relative submaximal workload.

[King Grub]

Mil Med. 1992 Feb;157(2):51-5. Effects of varying doses of smokeless tobacco at rest and during brief, high-intensity exercise.

To determine the influence of varying doses of smokeless tobacco (moist snuff) on resting heart rate and blood pressure and the performance of brief, high-intensity exercise, 12 snuff users were examined at rest and while performing Wingate Anaerobic Tests after taking six different doses of snuff. Snuff caused an increase in heart rate; the larger the dose, the larger the response. Increases in systolic and diastolic blood pressure also occurred but were similar at all doses. Performance of brief, high-intensity exercise was unaffected, so that there was no benefit for such activities (e.g., sprinting bases in base ball).

[King Grub]

J Appl Physiol. 1992 Feb;72(2):401-9. Metabolic effects of cigarette smoking.

The inverse relationship between cigarette smoking and body weight, a potent obstacle to stopping smoking, may be due in part to effects of smoking on increasing whole body metabolism. Studies examining chronic and acute metabolic effects of smoking, as well as its constituent nicotine, are reviewed. Evidence suggests the absence of a chronic effect; most studies indicate that smokers and nonsmokers have similar resting metabolic rates (RMR) and that RMR declines very little after smoking cessation. Although an acute effect due to smoking is apparent, its magnitude is inconsistent across studies, possibly because of variability in smoke exposure or nicotine intake. In smokers at rest, the acute effect of smoking (and nicotine intake) appears to be significant but small (less than 10% of RMR) and transient (less than or equal to 30 min). However, the specific situations in which smokers tend to smoke may mediate the magnitude of this effect, inasmuch as smoking during casual physical activity may enhance it while smoking after eating may reduce it. Sympathoadrenal activation by nicotine appears to be primarily responsible for the metabolic effect of smoking, but possible contributions from nonnicotine constituents of tobacco smoke and behavioral effects of inhaling may also be important. Improved understanding of these metabolic effects may lead to better prediction and control of weight gain after smoking cessation, thus increasing the likelihood of maintaining abstinence.

[King Grub]

Am J Clin Nutr. 1989 Sep;50(3):545-50. Acute effects of nicotine on resting metabolic rate in cigarette smokers.

The acute effects of nicotine on resting metabolic rate (RMR) were examined to identify a mechanism that may help explain the inverse association between smoking and body weight. Multiple administrations of two nicotine doses (moderate [15 micrograms/kg body wt] and low [7.5 micrograms/kg body wt]) and a placebo (0 micrograms) were presented to 18 male smokers via nasal-spray solution on three separate occasions while RMR was assessed by computerized open-circuit indirect calorimetry. Plasma nicotine levels confirmed the reliability of dosing. RMR increases of 6% above base line after both moderate and low doses were significantly greater than the 3% increase after the placebo. Subsequent examination of the effects of smoking a nonnicotine cigarette suggested that the small placebo effect was due to acute metabolic consequences of inhalation. These results confirm that intake of nicotine, isolated from tobacco smoke, significantly increases RMR in humans. However, the results also indicate that non-pharmacological, behavioral aspects of smoking may also contribute to acutely increasing RMR in smokers.

[King Grub]

Am J Clin Nutr. 1990 Aug;52(2):228-33. Metabolic effects of nicotine after consumption of a meal in smokers and nonsmokers.

The thermogenic effect of nicotine intake after calorie consumption was investigated to determine if nicotine influences metabolic response to a calorie challenge. Smokers and nonsmokers (10 males in each group), matched for body weight, age, and physical fitness, each participated in four sessions that involved consuming a liquid calorie load (4.77 kcal/kg body wt) or water, followed by nicotine (15 micrograms/kg body wt) or placebo via nasal spray every 20 min for 2 h. Energy expenditure was significantly increased above baseline resting metabolic rate (RMR) over the 2 h by nicotine alone (6.5% of RMR, p less than 0.01). However, the combined effect of nicotine after calorie load (20.1% of RMR, p less than 0.001) was not significantly greater than the effect of calorie load alone (18.4% of RMR, p less than 0.001). Smokers and nonsmokers did not differ in baseline RMR or in response to nicotine or calorie load. These results confirm the thermogenic effect of nicotine but suggest that the effect of nicotine after calorie consumption is less than additive.

[King Grub]

Physiol Behav. 1995 Jun;57(6):1097-102. Influence of aerobic fitness, activity level, and smoking history on the acute thermic effect of nicotine.

Individual variability in sensitivity to the acute thermic effects of nicotine has been difficult to study because of methodological limitations in the use of smoking to manipulate nicotine dose. This study examined the influence of smoking history, body composition, and, in a subset of subjects, aerobic fitness and physical activity on the thermic effect of nicotine using a measured-dose nasal spray procedure. Following overnight fasting and smoking abstinence, 38 healthy male smokers were intermittently administered a nicotine dose corrected for body weight (15 micrograms/kg, approx. 1.1 mg for average subject) or placebo on separate occasions in a within-subjects study. Indirect calorimetry was used to assess resting energy expenditure (REE) before and after dosing. Acute thermic response to nicotine ranged from -4.3 to +10.8 kcal/h (-5.4% to +12.6% of REE). Thermic response to nicotine was correlated significantly with aerobic fitness (r = 0.58, p < 0.005), physical activity (r = 0.44, p < 0.05), and number of pack-years of smoking (r = 0.43, p < 0.01). Thermic response was marginally correlated with percent body fat (r = -0.23, p < 0.10), but not with body weight (r = 0.04), percent of ideal weight for height (r = -0.10), or lean body mass (r = 0.05). These results indicate that male smokers higher in fitness and activity and with greater smoking exposure histories may experience greater increases in energy expenditure as a result of nicotine intake via smoking. Consequently, variability in these characteristics could help account for some of the variability in weight gain after stopping smoking.

[King Grub]

Am J Clin Nutr. 1994 Sep;60(3):312-9. Acute thermogenic effects of nicotine combined with caffeine during light physical activity in male and female smokers.

The thermogenic effects of nicotine and caffeine during physical activity compared with rest were examined in male and female smokers (n = 10 each). During eight sessions, nicotine (15 micrograms/kg) or placebo was given via measured-dose nasal spray intermittently after consumption of decaffeinated coffee with or without added caffeine (5 mg/kg), followed by assessment of energy expenditure by indirect calorimetry while subjects engaged in standardized, low-intensity cycle ergometer riding (activity) or remained at quiet rest. Results indicated significant thermogenic effects of nicotine and caffeine individually, with the combination of nicotine and caffeine producing additive effects. Expenditure attributable to nicotine, caffeine, or their combination was significantly enhanced during activity compared with rest, but only for males and not females. Plasma nicotine concentrations were influenced by activity and caffeine, but these pharmacokinetic changes did not appear to explain the differences in expenditure. These findings suggest a sex difference in thermogenic effects of nicotine and caffeine during casual physical activity and potentially explain some of the apparent individual variability in expenditure due to tobacco smoking.

[quinx]

Tack tack king grub!

frazze, jo idèn är väl lite *screwy* men man måste ju kolla :D

[oyeah]

Om jag inte snusar äter som fan. Så utan snus kan min deff hälsa hem. Kanske dumt o bli beroende av snus för en pytteliten skillnad? :D