Rökning i samband med styrketräning? effekter osv ?

[Nikejo]

Alla vet ju att rökning leder till minskad syreupptagning i musklerna och att detta givetvis påverkar träningen. Hur lång tid efter att man slutat röka kan man förvänta sig "normal" prestationsförmåga ?

tacksam för svar

[skaparn]

Bland många andra trådar i ämnet:

http://www.kolozzeum.com/kolozzeum/showthread.php?t=11275&highlight=r%F6kning

[Gastrocnemius]

Rökning påverkar myostatinet samt proteinsyntesen.

[Eddie Vedder]

Rökning påverkar myostatinet samt proteinsyntesen.

Ja det stämmer.:)

Smoking causes multiple organ dysfunction. The effect of smoking on skeletal muscle protein metabolism is unknown. We hypothesized that the rate of skeletal muscle protein synthesis is depressed in smokers compared with non-smokers. We studied eight smokers (20 cigarettes/day for 20 years) and eight non-smokers matched for sex (4 men and 4 women per group), age (65 ± 3 and 63 ± 3 yr, respectively; means ± SEM) and body mass index (25.9 ± 0.9 and 25.1 ± 1.2 kg/m2, respectively). Each subject underwent an intravenous infusion of stable isotope-labeled leucine in conjunction with blood and muscle tissue sampling to measure the mixed muscle protein fractional synthesis rate (FSR) and whole body leucine rate of appearance (Ra) in plasma (an index of whole body proteolysis), the expression of genes involved in the regulation of muscle mass (myostatin, a muscle growth inhibitor, and MAFBx and MuRF-1, which encode E3 ubiquitin ligases in the proteasome proteolytic pathway) and that for the inflammatory cytokine TNF- in muscle, and the concentration of inflammatory markers in plasma (C-reactive protein, TNF-, interleukin-6) which are associated with muscle wasting in other conditions. There were no differences between nonsmokers and smokers in plasma leucine concentration, leucine rate of appearance, and plasma concentrations of inflammatory markers, or TNF- mRNA in muscle, but muscle protein FSR was much less (0.037 ± 0.005 vs. 0.059 ± 0.005%/h, respectively, P = 0.004), and myostatin and MAFBx (but not MuRF-1) expression were much greater (by 33 and 45%, respectivley, P < 0.05) in the muscle of smokers than of nonsmokers. We conclude that smoking impairs the muscle protein synthesis process and increases the expression of genes associated with impaired muscle maintenance; smoking therefore likely increases the risk of sarcopenia.

http://ajpendo.physiology.org/cgi/content/full/293/3/E843

Smoking impairs muscle protein synthesis and increases the expression of myostatin and MAFbx in muscle. Am J Physiol Endocrinol Metab 293: E843-E848, 2007.

Och prestationsförmåga påverkas som sagt även den:

Abstract:
Background: Smoking is a major cause of cardiovascular disease and mortality. Smoking-related deaths in Greece account for 23%, whereas 41% of young Greeks are smokers, the highest percentage in Europe. The purpose of this study was to examine the effects of chronic smoking on the rate-pressure product and exercise tolerance in young, healthy male smokers.

Design and methods: Forty-two smokers and 51 nonsmokers were selected from a population of 543 students based on their age, sex, body mass index, physical fitness, smoking habit and health status. All participants were tested with the standard Bruce treadmill protocol. The rate-pressure product was obtained at rest and during exercise at a given submaximal workload. The evaluation of exercise tolerance was based on peak workload achieved and maximal exercise test duration.

Results: The smokers had a higher rate-pressure product at rest (P<0.001) due to their higher resting heart rate (P<0.001). Resting values of blood pressure did not differ significantly between the two groups. During exercise, smokers had a greater rate-pressure product (P<0.001), mainly due to their significantly higher systolic blood pressure (P=0.008). The smokers had a higher submaximal heart rate (P=0.005), but the differences in heart rate between groups were reduced for smokers during exercise when compared to rest. The smokers' exercise tolerance was impaired and their maximal exercise test duration time was significantly shorter (P<0.001).

Conclusions: Chronic smoking was found to affect young male smokers' cardiovascular fitness, impairing the economy and decreasing the capacity of their circulatory system.

http://www.jcardiovascularrisk.com/pt/re/ejcpr/fulltext.00149831-200710000-00008.htm;jsessionid=LDLQF8VyrvbwK402F9c3wvqx7ZTgt 0JljL3G2ZTK77zB9XQ4wS16!353761397!181195628!8091!-1

Effects of chronic smoking on exercise tolerance and on heart rate-systolic blood pressure product in young healthy adults. European Journal of Cardiovascular Prevention & Rehabilitation. 14(5):646-652, October 2007.

Något om prestationsförmåga efter att man slutat röka kunde jag tyvärr inte hitta.:(

[Elfsberg]

Ett som är säkert: det blir då inte sämre av att sluta! :)

[itsmylife]

Har hört att man stannar i vikten av rökning,vet dock om det inte är sant men det var en av alla orsakerna till att jag slutade!!

[Björkvedens mästare]

Att röka är onyttigt men såklart beror det på hur mycket man röker när det gäller skadligheten och nej man stannar inte i vikten av rökning ! Inte heller har du ständigt nersatt syresättnings förmåga utan bara när du har nikotin i dej, rök inte på 3 timmar och din syresättningsförmåga är som vanligt igen. Lungorna tar förvisso en del stryk.

[King Grub]

rök inte på 3 timmar och din syresättningsförmåga är som vanligt igen.

Om man förutsätter att rökarens kapacitet är det "vanliga" eller normen, så. Inte om man jämför med en icke-rökare.

Dessutom, var har du fått 3 timmar från?

The changes between before and after abstinence from smoking were as follows. The plasma cotinine had almost disappeared after the 7 d of abstinence from smoking. Pulmonary functions did not show a significant improvement.

Med Sci Sports Exerc. 2000 Feb;32(2):386-91.

[Alger]

Frågan är hur mycket det påvekrar en som inte tävlar exempelvis. Ganska mycket konditionsmässigt skulle jag tro. Men jag syftar mer på proteinsyntesen och det där. Jag röker inte själv så jag har inga praktiska erfarenheter.

[King Grub]

Frågan är hur mycket det påvekrar en som inte tävlar exempelvis. Ganska mycket konditionsmässigt skulle jag tro. Men jag syftar mer på proteinsyntesen och det där. Jag röker inte själv så jag har inga praktiska erfarenheter.

http://ajpendo.physiology.org/content/vol293/issue3/images/medium/zh10100750680001.gif

Mixed-muscle protein fractional synthesis rate (FSR). Values are means ± SE; n = 8 per group.

http://ajpendo.physiology.org/cgi/content/full/293/3/E843

[Eddie Vedder]

Men jag syftar mer på proteinsyntesen och det där.

Nu fick du ju ytterligare svar av King Grub men det stod ju faktiskt redan i det ena abstrakten jag citerade.

...muscle protein FSR was much less (0.037 ± 0.005 vs. 0.059 ± 0.005%/h, respectively, P = 0.004), and myostatin and MAFBx (but not MuRF-1) expression were much greater (by 33 and 45%, respectivley, P < 0.05) in the muscle of smokers than of nonsmokers. We conclude that smoking impairs the muscle protein synthesis process and increases the expression of genes associated with impaired muscle maintenance; smoking therefore likely increases the risk of sarcopenia.

Jag röker inte själv så jag har inga praktiska erfarenheter.

Oavsett om du hade rökt tror jag nog inte du hade direkt känt av en försämrad muskelproteinsyntes i vilket fall.;)